The brain on psilocybin.
Mechanism work explaining how a single dose can outlast the dose. Several of these are animal or cell studies, marked as such, and none of them are evidence of a clinical benefit on their own.
7 studies · 3 early · 4 moderatePsilocybin disrupted brain communication far more than a stimulant
Brains became less compartmentalised, and moods lifted with them
Activity went down, not up
Psychedelics grew neurons in a dish, a fly and a rat
Why serotonin itself does not do this
Blocking one receptor switched the whole experience off
This is where the word neuroplasticity comes from in psilocybin marketing. Worth reading the actual studies, because most of them were done in mice.
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